Figure 1From: Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammationRepresentative multiprobe ribonuclease protection assay results indicating selective increase in certain cytokine mRNAs in G93A-SOD1 astrocyte cultures, either in the absence of deliberate stimulation (basal condition) or after 4 hours treatment with recombinant murine IFNγ (50 U/mL), TNFα (40 ng/mL) or both. Each lane represents pooled mRNA from at 6 wells of cells.Back to article page