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Figure 3 | Journal of Neuroinflammation

Figure 3

From: Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation

Figure 3

iNOS protein expression and NO2- formation in cultured nontransgenic or G93A-SOD1+ astrocytes in the basal state and after 48 hours stimulation with recombinant murine TNFα (40 ng/mL) plus IFNγ (50 U/mL). Bars represent mean ± SD from 6 wells of cells in a typical experiment; * p < 0.05 for stimulated G93A-SOD1+ cells relative to correspondingly treated nontransgenic cells, by two-tailed t-test.

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Journal of Neuroinflammation

ISSN: 1742-2094