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Table 1 Acute administration of HMGB1 (0.65 μg/ml) elicits an intracellular calcium flux in primary sensory neurons

From: The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain

  Naïve
  Capsaicin- sensitive neurons Non-capsaicin-sensitive neurons
HMGB1 70% (31/44) 19% (26/136)