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Table 1 Acute administration of HMGB1 (0.65 μg/ml) elicits an intracellular calcium flux in primary sensory neurons

From: The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain

 

Naïve

 

Capsaicin- sensitive neurons

Non-capsaicin-sensitive neurons

HMGB1

70% (31/44)

19% (26/136)

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Journal of Neuroinflammation

ISSN: 1742-2094