Fig. 5
From: Ccr2 deletion dissociates cavity size and tau pathology after mild traumatic brain injury
Assessment of tau phosphorylation by Western blot. Protein lysates from the cortex (a) or hippocampus (b) of treatment-naïve, sham (surgery only), or mild injured control or Ccr2 RFP/RFP mice were probed for pTau (AT8 antibody, green). GAPDH was used as loading control (red). Mild injury induced a visible increase on the ipsilateral side of the cortex and hippocampus in Ccr2 RFP/RFP mice