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Fig. 2 | Journal of Neuroinflammation

Fig. 2

From: Increased occurrence of protein kinase CK2 in astrocytes in Alzheimer’s disease pathology

Fig. 2

Immunohistochemical detection of CK2α/α’ in control and AD brain. Shown are representative immunohistochemical stainings for CK2α/α’ of the mid-temporal cortex and hippocampus of a control (af, Braak stage 0, Table 1C case #25) and two AD cases (Braak stage VI, Table 1C cases gi #35 and jl #36). a CK2α/α’ immunoreactivity in the grey matter of the mid-temporal cortex in a control case. b CK2α/α’ immunoreactivity in the subpial layer of the mid-temporal cortex in a control cases. c Detailed picture of CK2α/α’ immunoreactivity in astrocytes around a blood vessel in the grey matter of the mid-temporal cortex in a control case. d Overview of CK2α/α’ immunoreactivity in astrocytes in the white matter (temporal cortex) of a control case. e Detailed picture of CK2α/α’ immunoreactivity in the white matter of a control case. f CK2α/α’ immunoreactivity in the CA1 region of the hippocampus of a control case. g CK2α/α’ immunoreactivity in the grey matter of the mid-temporal cortex in an AD case. h, i Detailed pictures of clusters of astrocytes with CK2α/α’ immunoreactivity in the grey matter of the mid-temporal cortex in an AD case. j Overview of CK2α/α’ immunoreactivity in the white matter (temporal cortex) of an AD case. k Detailed picture of CK2α/α’ immunoreactivity in astrocytes in the white matter of an AD case. l CK2α/α’ immunoreactivity in the CA1 region of the hippocampus of an AD case. Immunohistochemical detection was performed using DAB (brown) and nuclei were stained with haematoxylin (blue). Scale bars a, b, d, g and j 200 μm; f and l 100 μm; c, e, h, i and k 50 μm

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