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Fig. 1 | Journal of Neuroinflammation

Fig. 1

From: The Carbon monoxide releasing molecule ALF-186 mediates anti-inflammatory and neuroprotective effects via the soluble guanylate cyclase ß1 in rats’ retinal ganglion cells after ischemia and reperfusion injury

Fig. 1

Influence of ALF-186 treatment on vital retinal ganglion cell count after ischemia reperfusion injury (IRI). a Quantification of retinal ganglion cell density (cells/mm2, data are mean ± SD, n = 8, IRI vs. IRI + ALF-186 0 h and 3 h, *** = p < 0.001 and IRI + ALF-186 0 h vs. IRI + ALF-186 + ODQ, * = p < 0.05; n.s. = not significant). b Representative flat mount images (n = 8) of fluorogold-labeled retinal ganglion cells 7 days after IRI, immediate and delayed (3 h) ALF-186 treatment, inactivated ALF-186 treatment and SGC inhibition with ODQ

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Journal of Neuroinflammation

ISSN: 1742-2094