Fig. 4From: Aggf1 attenuates neuroinflammation and BBB disruption via PI3K/Akt/NF-κB pathway after subarachnoid hemorrhage in ratsExogenous Aggf1 alleviated neutrophil infiltration and inflammatory molecule expression at 24 h subarachnoid hemorrhage (SAH). a Immunofluorescence revealed that treatment with rh-Aggf1 reduced the number of MPO-positive cells in the ipsilateral cortex, n = 3 per group, scale bar = 100 μm. b, c Relative fold changes of inflammatory cytokines (TNF-α and IL-1β) after exogenous Aggf1 treatment, n = 6. #P < 0.05 vs sham; *P < 0.05 vs vehicle. MPO, myeloperoxidaseBack to article page