Fig. 5From: Normalization of magnesium deficiency attenuated mechanical allodynia, depressive-like behaviors, and memory deficits associated with cyclophosphamide-induced cystitis by inhibiting TNF-α/NF-κB signaling in female ratsTNF-α, p-p65, and IL-1β all co-localize with NeuN in the SDH of CYP-induced cystitis rats. Photographs of the double immunofluorescence staining indicate that TNF-α, p-p65 (a principal transcriptional regulator of the activation of NF-kB pathway), and IL-1β were only co-localized with NeuN-labeled neurons but not with GFAP-labeled astrocytes or OX-42-labeled microglia in the SDH of cystitis animalsBack to article page