Fig. 3From: Molecular mechanisms underlying the actions of arachidonic acid-derived prostaglandins on peripheral nociceptionPro-nociceptive effector molecules that contribute to pain exacerbation by PGs in somatosensory neurons. The functions of diverse ion channels, transporters, and metabotropic receptors are altered by the signal transductions described in Fig. 2, eventually promoting the electrical excitability, neurogenic inflammation, and neuritogenesis of somatosensory neuronsBack to article page