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Fig. 7 | Journal of Neuroinflammation

Fig. 7

From: Recruitment of γδ T cells to the lesion via the CCL2/CCR2 signaling after spinal cord injury

Fig. 7

TCRδ−/− mice were reconstituted with γδ T cells from CCR2−/− and WT mice. a BMS score of WT and CCR2−/− γδ T cells reconstituted mice at different time points after spinal cord injury (SCI) (n = 8 mice/per group). b Max contact area of WT and CCR2−/− γδ T cells reconstituted mice at 6wk after SCI (n = 8 mice/per group). c Regularity index of WT and CCR2−/− γδ T cells reconstituted mice at 6 weeks after SCI (n = 8 mice/per group). d Cadence of WT and CCR2−/− γδ T cells reconstituted mice at 6wk after SCI (n = 8 mice/per group). e Motor-evoked potential (MEP) recordings from WT and CCR2−/− γδ T cells reconstituted mice at 6 weeks post-surgery (n = 8 mice/per group). f Spinal sections from WT and CCR2−/− γδ T cells reconstituted mice after 1 day post-SCI were immunostained with anti-TCRγδ (green, a maker for γδ T cell) and the corresponding static histogram of percent of γδ T cell (γδ T cell/total cell) (n = 6 mice/per group). g Survival of motor neurons immunostained with Nissl staining in cross-section of lesion epicenter at 6 weeks after SCI (n = 6 mice/per group). *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001

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