Fig. 9From: Inhibition of CCR2 attenuates neuroinflammation and neuronal apoptosis after subarachnoid hemorrhage through the PI3K/Akt pathwaySchematic diagram for CCR2 following SAH. CCR2 was received by bioinformatics analysis after SAH. It induces early brain injury by regulating neuroinflammation and neuronal apoptosis and is positively correlated with clinical outcome of SAH patientsBack to article page