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  1. Though accumulating evidence suggests that microglia, resident macrophages in the retina, and bone marrow-derived macrophages can cause retinal inflammation which accelerates photoreceptor cell death, the deta...

    Authors: Hideo Kohno, Hideto Koso, Kiichiro Okano, Thomas R. Sundermeier, Saburo Saito, Sumiko Watanabe, Hiroshi Tsuneoka and Tsutomu Sakai
    Citation: Journal of Neuroinflammation 2015 12:188
  2. The family of 14-3-3 proteins plays an important role in the regulation of cell survival and death. Here, we investigate the role of the 14-3-3 gamma (14-3-3 γ) subunit for glial responses in autoimmune demyel...

    Authors: De-Hyung Lee, Petra Steinacker, Silvia Seubert, Tanja Turnescu, Arthur Melms, Arndt Manzel, Markus Otto and Ralf A. Linker
    Citation: Journal of Neuroinflammation 2015 12:187
  3. Mild traumatic brain injury (mTBI) is an all too common occurrence that exacts significant personal and societal costs. The pathophysiology of mTBI is complex, with reports routinely correlating diffuse axonal...

    Authors: Audrey D. Lafrenaye, Masaki Todani, Susan A. Walker and John T. Povlishock
    Citation: Journal of Neuroinflammation 2015 12:186
  4. Astrocytes expressing the aquaporin-4 water channel are a primary target of pathogenic, disease-specific immunoglobulins (IgG) found in patients with neuromyelitis optica (NMO). Immunopathological analyses of ...

    Authors: Margaret E. Walker-Caulfield, Yong Guo, Renee K. Johnson, Christina B. McCarthy, Patrick D. Fitz-Gibbon, Claudia F. Lucchinetti and Charles L. Howe
    Citation: Journal of Neuroinflammation 2015 12:185
  5. Fingolimod (FTY720) is the first sphingosine-1-phosphate (S1P) receptor modulator approved for the treatment of multiple sclerosis. The phosphorylated active metabolite FTY720-phosphate (FTY-P) interferes with...

    Authors: Franziska S. Hoffmann, Johann Hofereiter, Heike Rübsamen, Johannes Melms, Sigrid Schwarz, Hans Faber, Peter Weber, Benno Pütz, Verena Loleit, Frank Weber, Reinhard Hohlfeld, Edgar Meinl and Markus Krumbholz
    Citation: Journal of Neuroinflammation 2015 12:184
  6. Liver failure in experimental animals or in human cirrhosis elicits neuroinflammation. Prolyl oligopeptidase (PREP) has been implicated in neuroinflammatory events in neurodegenerative diseases: PREP protein l...

    Authors: Jofre Tenorio-Laranga, Carmina Montoliu, Amparo Urios, Vicente Hernandez-Rabaza, Hanan Ahabrach, J. Arturo García-Horsman and Vicente Felipo
    Citation: Journal of Neuroinflammation 2015 12:183
  7. Sepsis-associated encephalopathy (SAE) is a diffuse brain dysfunction caused by many pathological events, including neuroinflammation and oxidative stress damage. Increasing evidence suggests that parvalbumin ...

    Authors: Mu-Huo Ji, Li-Li Qiu, Hui Tang, Ling-Sha Ju, Xiao-Ru Sun, Hui Zhang, Min Jia, Zhi-Yi Zuo, Jin-Chun Shen and Jian-Jun Yang
    Citation: Journal of Neuroinflammation 2015 12:182
  8. Non-infectious inflammatory diseases of the canine central nervous system (CNS) are common idiopathic disorders grouped under the term meningoencephalomyelitis of unknown origin (MUO). Ante mortem diagnosis is...

    Authors: Offer Zeira, Nimrod Asiag, Marina Aralla, Erica Ghezzi, Letizia Pettinari, Laura Martinelli, Daniele Zahirpour, Maria Pia Dumas, Davide Lupi, Simone Scaccia, Martin Konar and Carlo Cantile
    Citation: Journal of Neuroinflammation 2015 12:181
  9. Preterm infants are especially vulnerable to infection-induced white matter injury, associated with cerebral palsy, cognitive and psychomotor impairment, and other adverse neurological outcomes. The etiology o...

    Authors: Chen Jin, Irene Londono, Carina Mallard and Gregory A. Lodygensky
    Citation: Journal of Neuroinflammation 2015 12:180
  10. It is not clear how invading autoreactive T cells initiate the pathogenic process inside the diseased organ in T cell-mediated organ-specific autoimmune disease. In experimental autoimmune uveitis (EAU) induce...

    Authors: Guomin Jiang, Yunsong Wang, Juan Yun, Amir Reza Hajrasouliha, Yuan Zhao, Deming Sun, Henry J Kaplan and Hui Shao
    Citation: Journal of Neuroinflammation 2015 12:179
  11. West Nile virus (WNV) is a neurotropic flavivirus that has emerged globally as a significant cause of viral encephalitis in humans. Herein, we investigated the immunological responses induced by two phylogenet...

    Authors: Mukesh Kumar, Kelsey Roe, Maile O’Connell and Vivek R. Nerurkar
    Citation: Journal of Neuroinflammation 2015 12:178
  12. Pathological features of Alzheimer’s disease (AD) include aggregation of amyloid beta (Aβ) and tau protein. Macrophage migration inhibitory factor (MIF), a proinflammatory cytokine, has been implicated in the ...

    Authors: Shu-Qin Li, Yang Yu, Jin-Zhao Han, Ding Wang, Jin Liu, Feng Qian, Guo-Huang Fan, Richard Bucala and Richard D. Ye
    Citation: Journal of Neuroinflammation 2015 12:177
  13. Neuroinflammation—astrogliosis, microglial activation, and changes in cytokine signaling—is a prominent feature of neurodegenerative disorders. Glaucoma is a group of chronic neurodegenerative conditions that ...

    Authors: Gina N. Wilson, Denise M. Inman, Christine M. Dengler-Crish, Matthew A. Smith and Samuel D. Crish
    Citation: Journal of Neuroinflammation 2015 12:176

    The Erratum to this article has been published in Journal of Neuroinflammation 2015 12:191

  14. Acute hemorrhagic leukoencephalitis is a fulminant demyelinating disease and commonly considered as a rare and severe variant of acute disseminated encephalomyelitis. Here, we report the clinical, magnetic res...

    Authors: Özlem Yildiz, Refik Pul, Peter Raab, Christian Hartmann, Thomas Skripuletz and Martin Stangel
    Citation: Journal of Neuroinflammation 2015 12:175
  15. Aging is associated with a decline in stem cell proliferation that is thought to be a result of dysregulated signaling in the neurogenic niche. This results in a diminished and less efficient pool of progenito...

    Authors: Antwoine Flowers, Jea-Young Lee, Sandra Acosta, Charles Hudson, Brent Small, Cyndy D. Sanberg, Paula C. Bickford and Bethany Grimmig
    Citation: Journal of Neuroinflammation 2015 12:174

    The Erratum to this article has been published in Journal of Neuroinflammation 2016 13:229

  16. Experimental autoimmune encephalomyelitis (EAE) is a model of inflammatory demyelinating diseases mediated by different types of leukocytes. How these cells communicate with each other to orchestrate autoimmun...

    Authors: Lusine Bozoyan, Aline Dumas, Alexandre Patenaude and Luc Vallières
    Citation: Journal of Neuroinflammation 2015 12:173
  17. Spinal cord injury (SCI) results in the activation of the NADPH oxidase (NOX) enzyme, inducing production of reactive oxygen species (ROS). We hypothesized that the NOX2 isoform plays an integral role in post-...

    Authors: Guzal Khayrullina, Sara Bermudez and Kimberly R. Byrnes
    Citation: Journal of Neuroinflammation 2015 12:172
  18. HIV-associated neuroinflammation is believed to be a major contributing factor in the development of HIV-associated neurocognitive disorders (HAND). In this study, we used micropositron emission tomography (PE...

    Authors: Dianne E. Lee, Xuyi Yue, Wael G. Ibrahim, Margaret R. Lentz, Kristin L. Peterson, Elaine M. Jagoda, Michael Kassiou, Dragan Maric, William C. Reid and Dima A. Hammoud
    Citation: Journal of Neuroinflammation 2015 12:171
  19. Research focus in neuro-oncology has shifted in the last decades towards the exploration of tumor infiltration by a variety of immune cells and their products. T cells, macrophages, B cells, and mast cells (MC...

    Authors: Stavros Polyzoidis, Triantafyllia Koletsa, Smaro Panagiotidou, Keyoumars Ashkan and Theoharis C. Theoharides
    Citation: Journal of Neuroinflammation 2015 12:170
  20. Experimental autoimmune encephalomyelitis (EAE) in the common marmoset monkey (Callithrix jacchus) is a relevant preclinical model for translational research into immunopathogenic mechanisms operating in multiple...

    Authors: S. Anwar Jagessar, Nicole Heijmans, Erwin L A Blezer, Jan Bauer, Robert Weissert and Bert A. ‘t Hart
    Citation: Journal of Neuroinflammation 2015 12:169
  21. Serological testing for anti-neural autoantibodies is important in patients presenting with idiopathic cerebellar ataxia, since these autoantibodies may indicate cancer, determine treatment and predict prognos...

    Authors: S. Jarius and B. Wildemann
    Citation: Journal of Neuroinflammation 2015 12:168
  22. Serological testing for anti-neural autoantibodies is important in patients presenting with idiopathic cerebellar ataxia, since these autoantibodies may indicate cancer, determine treatment and predict prognos...

    Authors: S. Jarius and B. Wildemann
    Citation: Journal of Neuroinflammation 2015 12:167
  23. Serological testing for anti-neural autoantibodies is important in patients presenting with idiopathic cerebellar ataxia, since these autoantibodies may indicate cancer, determine treatment and predict prognos...

    Authors: S. Jarius and B. Wildemann
    Citation: Journal of Neuroinflammation 2015 12:166
  24. Neuroinflammation is considered a risk factor for impairments in neuronal function and cognition that arise with trauma, infection, and/or disease. IL-17A has been determined to be involved in neurodegenerativ...

    Authors: Jie Sun, Susu Zhang, Xiang Zhang, Xiaobao Zhang, Hongquan Dong and Yanning Qian
    Citation: Journal of Neuroinflammation 2015 12:165
  25. Authors: Sanish Sathyan, Linda V. Koshy, Lekshmy Srinivas, H. V. Easwer, S. Premkumar, Suresh Nair, R. N. Bhattacharya, Jacob P. Alapatt and Moinak Banerjee
    Citation: Journal of Neuroinflammation 2015 12:164

    The original article was published in Journal of Neuroinflammation 2015 12:135

  26. Evidence from clinical studies and animal models show that inflammation can lead to the development of depression. Macrophage migration inhibitory factor (MIF) is an important multifunctional cytokine that is ...

    Authors: Cecilie Bay-Richter, Shorena Janelidze, Analise Sauro, Richard Bucala, Jack Lipton, Tomas Deierborg and Lena Brundin
    Citation: Journal of Neuroinflammation 2015 12:163
  27. Status epilepticus (SE) is a severe condition that may lead to hippocampal cell loss and epileptogenesis. Some of the mechanisms associated with SE-induced cell death are excitotoxicity, neuroinflammation, and...

    Authors: Beatriz O. Amorim, Luciene Covolan, Elenn Ferreira, José Geraldo Brito, Diego P. Nunes, David G. de Morais, José N. Nobrega, Antonio M. Rodrigues, Antonio Carlos G. deAlmeida and Clement Hamani
    Citation: Journal of Neuroinflammation 2015 12:162
  28. The NG2 proteoglycan is expressed by several cell types in demyelinated lesions and has important effects on the biology of these cells. Here we determine the cell-type-specific roles of NG2 in the oligodendro...

    Authors: Karolina Kucharova and William B. Stallcup
    Citation: Journal of Neuroinflammation 2015 12:161
  29. Fetal alcohol exposure is a leading cause of preventable birth defects, yet drinking during pregnancy remains prevalent worldwide. Studies suggest that activation of the neuroimmune system plays a role in the ...

    Authors: Lauren A. Topper, Brian C. Baculis and C. Fernando Valenzuela
    Citation: Journal of Neuroinflammation 2015 12:160
  30. Pilakka-Kanthikeel et al. recently reported higher levels of the retroviral restriction factor sterile alpha motif and histidine/aspartic acid domain-containing protein 1 (SAMHD1) in astrocytes than in microgl...

    Authors: Kenneth W. Witwer, Erin L. Buchanan, Stephanie L. Myers and Melissa A. McAlexander
    Citation: Journal of Neuroinflammation 2015 12:159
  31. Mechanical pain hypersensitivity associated with physical trauma to peripheral nerve depends on T-helper (Th) cells expressing the algesic cytokine, interleukin (IL)-17A. Fibronectin (FN) isoform alternatively...

    Authors: Huaqing Liu, Jennifer Dolkas, Khan Hoang, Mila Angert, Andrei V. Chernov, Albert G. Remacle, Sergey A. Shiryaev, Alex Y. Strongin, Tasuku Nishihara and Veronica I. Shubayev
    Citation: Journal of Neuroinflammation 2015 12:158
  32. Multiple sclerosis (MS) is an autoimmune inflammatory and neurodegenerative disease of the central nervous system (CNS). It is widely accepted that inflammatory cells play major roles in the pathogenesis of MS...

    Authors: Yohannes Haile, Katia Carmine-Simmen, Camille Olechowski, Bradley Kerr, R. Chris Bleackley and Fabrizio Giuliani
    Citation: Journal of Neuroinflammation 2015 12:157
  33. Mounting evidence has indicated that high-mobility group box 1 (HMGB1) is involved in cell activation and migration. Our previous study demonstrated that methamphetamine mediates activation of astrocytes via s...

    Authors: Yuan Zhang, Tiebing Zhu, Xiaotian Zhang, Jie Chao, Gang Hu and Honghong Yao
    Citation: Journal of Neuroinflammation 2015 12:156
  34. Neuroendoscopy is an innovative technique for neurosurgery that can nonetheless result in traumatic brain injury. The accompanying neuroinflammation may lead to secondary tissue damage, which is the major caus...

    Authors: Yang Xia, Liang Kong, Yingjia Yao, Yanan Jiao, Jie Song, Zhenyu Tao, Zhong You and Jingxian Yang
    Citation: Journal of Neuroinflammation 2015 12:155
  35. Neuroinflammation is an important secondary mechanism that is a key mediator of the long-term consequences of neuronal injury that occur in traumatic brain injury (TBI). Microglia are highly plastic cells with...

    Authors: Heidi Y. Febinger, Hannah E. Thomasy, Maria N. Pavlova, Kristyn M. Ringgold, Paulien R. Barf, Amrita M. George, Jenna N. Grillo, Adam D. Bachstetter, Jenny A. Garcia, Astrid E. Cardona, Mark R. Opp and Carmelina Gemma
    Citation: Journal of Neuroinflammation 2015 12:154
  36. Senile plaques consisting of amyloid-beta (Aβ) are the major pathological hallmark of Alzheimer’s disease (AD) and have been the primary therapeutic target. Immunotherapies, which are designed to remove brain ...

    Authors: Yu-Hui Liu, Xian-Le Bu, Chun-Rong Liang, Ye-Ran Wang, Tao Zhang, Shu-Sheng Jiao, Fan Zeng, Xiu-Qing Yao, Hua-Dong Zhou, Juan Deng and Yan-Jiang Wang
    Citation: Journal of Neuroinflammation 2015 12:153
  37. In brain inflammatory diseases, axonal damage is one of the most critical steps in the cascade that leads to permanent disability. Thus, identifying the initial events triggered by inflammation or oxidative st...

    Authors: Oihana Errea, Beatriz Moreno, Alba Gonzalez-Franquesa, Pablo M. Garcia-Roves and Pablo Villoslada
    Citation: Journal of Neuroinflammation 2015 12:152

    The Correction to this article has been published in Journal of Neuroinflammation 2017 14:206

  38. Spinal central sensitization is an important process in the generation and maintenance of visceral hypersensitivity. The release of brain-derived neurotrophic factor (BDNF) from the primary afferent neurons to...

    Authors: Miao Liu, Jarren C Kay, Shanwei Shen and Li-Ya Qiao
    Citation: Journal of Neuroinflammation 2015 12:151
  39. Alzheimer’s disease (AD) is the most common neurodegenerative disease characterized by neuronal loss due to amyloid beta aggregations, neurofibrillary tangles, and prominent neuroinflammation. Recently, interf...

    Authors: Christine Landlinger, Lisa Oberleitner, Petra Gruber, Birgit Noiges, Kristyna Yatsyk, Radmila Santic, Markus Mandler and Guenther Staffler
    Citation: Journal of Neuroinflammation 2015 12:150
  40. We compared adjunctive treatment with placebo, dexamethasone, anti-C5 antibodies, and the combination of dexamethasone plus anti-C5 antibodies in experimental pneumococcal meningitis.

    Authors: E. Soemirien Kasanmoentalib, Mercedes Valls Seron, B. Paul Morgan, Matthijs C. Brouwer and Diederik van de Beek
    Citation: Journal of Neuroinflammation 2015 12:149
  41. Intravenous immunoglobulin (IVIg) is an effective treatment in chronic inflammatory demyelinating polyneuropathy (CIDP). In most patients, the optimal IVIg dose and regime is unknown. Polyvalent immunoglobulin...

    Authors: Christian Ritter, Ilja Bobylev and Helmar C. Lehmann
    Citation: Journal of Neuroinflammation 2015 12:148
  42. Neuroinflammation occurs in insulted regions of the brain and may be due to reactive oxygen species (ROS), nitric oxide (NO), cytokines, and chemokines produced by activated glia. Excessive production of neuro...

    Authors: Ya-Ni Huang, Yi-Jung Ho, Chien-Cheng Lai, Chien-Tsai Chiu and Jia-Yi Wang
    Citation: Journal of Neuroinflammation 2015 12:147
  43. The purpose of this research was to validate the low expression of L-selectin (CD62L) in natalizumab (NTZ)-treated patients. CD62L is involved in rolling and transmigration of leukocyte cells. A correlation be...

    Authors: Michela Spadaro, Marzia Caldano, Fabiana Marnetto, Alessandra Lugaresi and Antonio Bertolotto
    Citation: Journal of Neuroinflammation 2015 12:146
  44. It has recently become evident that activating/inhibitory cell surface immune receptors play a critical role in regulating immune and inflammatory processes in the central nervous system (CNS). The immunorecep...

    Authors: Hugo Peluffo, Patricia Solari-Saquieres, Maria Luciana Negro-Demontel, Isaac Francos-Quijorna, Xavier Navarro, Ruben López-Vales, Joan Sayós and Natalia Lago
    Citation: Journal of Neuroinflammation 2015 12:145
  45. The chemokine interleukin-8 (IL-8) and its receptor CXCR2 contribute to chemotactic responses in Alzheimer’s disease (AD); however, properties of the ligand and receptor have not been characterized in animal m...

    Authors: Jae K Ryu, T Cho, Hyun B Choi, N Jantaratnotai and James G McLarnon
    Citation: Journal of Neuroinflammation 2015 12:144
  46. NOD-like receptors (Nlrs) are key regulators of immune responses during infection and autoimmunity. A subset of Nlrs assembles inflammasomes, molecular platforms that are activated in response to endogenous da...

    Authors: Elke Ydens, Dieter Demon, Guillaume Lornet, Vicky De Winter, Vincent Timmerman, Mohamed Lamkanfi and Sophie Janssens
    Citation: Journal of Neuroinflammation 2015 12:143
  47. Diabetic retinopathy (DR) is a common complication of diabetes and has been recognized as a vascular dysfunction leading to blindness in working-age adults. It becomes increasingly clear that neural cells in r...

    Authors: Ying Yu, Hui Chen and Shao Bo Su
    Citation: Journal of Neuroinflammation 2015 12:141
  48. Obesity increases the risk for ischaemic stroke and is associated with worse outcome clinically and experimentally. Most experimental studies have used genetic models of obesity. Here, a more clinically releva...

    Authors: Samaneh Maysami, Michael J. Haley, Natalia Gorenkova, Siddharth Krishnan, Barry W McColl and Catherine B Lawrence
    Citation: Journal of Neuroinflammation 2015 12:140
  49. Microglia are dependent upon colony-stimulating factor 1 receptor (CSF1R) signaling for their survival in the adult brain, with administration of the dual CSF1R/c-kit inhibitor PLX3397 leading to the near-comp...

    Authors: Nabil N. Dagher, Allison R. Najafi, Kara M. Neely Kayala, Monica R. P. Elmore, Terra E. White, Rodrigo Medeiros, Brian L. West and Kim N. Green
    Citation: Journal of Neuroinflammation 2015 12:139

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