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Figure 2 | Journal of Neuroinflammation

Figure 2

From: Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation

Figure 2

Comparison of basal and cytokine-stimulated PGE2 and LTB4 production by nontransgenic primary mouse astrocytes, G93A-SOD1 mouse astrocytes, or wild type human SOD1-expressing mouse astrocytes. Insets show western blot analysis of basal COX-II and 5-LOX protein expression. Data bars indicate mean ± SD of 6 wells of cells from a typical experiment. p < 0.05 overall by ANOVA; * indicates specific difference between nontransgenic and G93A-SOD1 cultures assessed by Bonferroni post-hoc tests.

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Journal of Neuroinflammation

ISSN: 1742-2094