Skip to main content
Fig. 2 | Journal of Neuroinflammation

Fig. 2

From: Upregulation of neuronal zinc finger protein A20 expression is required for electroacupuncture to attenuate the cerebral inflammatory injury mediated by the nuclear factor-kB signaling pathway in cerebral ischemia/reperfusion rats

Fig. 2

The spatial distribution and cell-type location of A20 in the rat brain after I/R. Brain sections of the ischemia/reperfusion cortex of rats in the MCAO + EA group 24 h after reperfusion. Sections were stained with double immunofluorescence labeling (a–d), which showed the upregulated expression of A20 protein (red) in neurons (green, identified using NeuN). e–h Very few of A20-positive (red) astrocytes (green, identified using GFAP) were observed. i–l A20 (red) was barely presented in microglial cells (green, identified using iba-I). Scale bar = 50 μm. 1–3 were the magnified parts from the merged images of d, h, l which more clearly showed the location relationship between A20 and neurons, astrocytes, and microglia, respectively. Scale bar = 25 μm

Back to article page

Journal of Neuroinflammation

ISSN: 1742-2094