Fig. 6From: The neuroprotective mechanism of sevoflurane in rats with traumatic brain injury via FGF2Schematic diagram of the mechanism by which Sevo affects TBI via the FGF2/EZH2/HES1 axis. Sevo upregulates FGF2 to elevate EZH2 expression, promote the methylation of the promoter of HES1, and inhibit the transcription of HES1, thereby inhibiting neuronal apoptosis and autophagy, and ultimately promoting the repair of neurological function in TBI ratsBack to article page