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Correction to: Exosomes derived from atorvastatin-modified bone marrow dendritic cells ameliorate experimental autoimmune myasthenia gravis by up-regulated levels of IDO/Treg and partly dependent on FasL/Fas pathway

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The original article was published in Journal of Neuroinflammation 2016 13:8

Correction to: J Neuroinflammation

https://doi.org/10.1186/s12974-016-0475-0

After the publication of the original article [1], it came to the authors’ attention that there was an error in the originally published version of Fig. 5b. The image of CD4+CD25+ T cells of the statin-Dex group was unintentionally replaced with the image of CD4+CD25+ T cells from the control group. The correct version of Fig. 5b is published in this Erratum.

Fig. 5
figure1

Statin-Dex treatment increase the number of CD4+Foxp3+ T cells in lymphocytes. Expressions of CD4+CD25+ T cells and CD4+Foxp3+ T cells among lymph node MNC in statin-Dex group, control-Dex group, and control group were detected by FACS. The results showed that statin-Dex treatment increased the percentage of CD4+Foxp3+ T cells among lymph node MNC when compared with control-Dex and PBS treatments, while there was no difference for the percentage of CD4+CD25+ T cells. Meanwhile, we did not observe difference in the percentages of CD4+CD25+ T cells and CD4+Foxp3+ T cells between control-Dex group and control group (a, b). The results are expressed as mean ± SD (n = 5 rats per group) (*p < 0.05)

Reference

  1. 1.

    Li X-L, Li H, Zhang M, Xu H, Yue L-T, Zhang X-X, Wang S, Wang C-C, Li Y-B, Dou Y-C, Duan R-S. Exosomes derived from atorvastatin-modified bone marrow dendritic cells ameliorate experimental autoimmune myasthenia gravis by up-regulated levels of IDO/Treg and partly dependent on FasL/Fas pathway. J Neuroinflammation. 2016;13:8 https://doi.org/10.1186/s12974-016-0475-0.

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Correspondence to Rui-Sheng Duan.

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Li, X., Li, H., Zhang, M. et al. Correction to: Exosomes derived from atorvastatin-modified bone marrow dendritic cells ameliorate experimental autoimmune myasthenia gravis by up-regulated levels of IDO/Treg and partly dependent on FasL/Fas pathway. J Neuroinflammation 16, 119 (2019) doi:10.1186/s12974-019-1503-7

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