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  1. Overproduction of proinflammatory cytokines from activated microglia has been implicated as an important contributor to pathophysiology progression in both acute and chronic neurodegenerative diseases. Therefo...

    Authors: Adam D Bachstetter, Bin Xing, Lucia de Almeida, Edgardo R Dimayuga, D Martin Watterson and Linda J Van Eldik
    Citation: Journal of Neuroinflammation 2011 8:79
  2. Monocyte chemoattractant protein (CCL2/MCP-1) is a chemokine that attracts cells involved in the immune/inflammatory response. As microglia are one of the main cell types sustaining inflammation in brain, we p...

    Authors: Ara E Hinojosa, Borja Garcia-Bueno, Juan C Leza and Jose LM Madrigal
    Citation: Journal of Neuroinflammation 2011 8:77
  3. Macrophage migration inhibitory factor (MIF) has been proposed to play a detrimental role in stroke. We recently showed that MIF promotes neuronal death and aggravates neurological deficits during the first we...

    Authors: Ana R Inácio, Richard Bucala and Tomas Deierborg
    Citation: Journal of Neuroinflammation 2011 8:75
  4. Granulocyte colony stimulating factor (GCSF) is protective in animal models of various neurodegenerative diseases. We investigated whether pegfilgrastim, GCSF with sustained action, is protective in a mouse mo...

    Authors: Eveliina Pollari, Ekaterina Savchenko, Merja Jaronen, Katja Kanninen, Tarja Malm, Sara Wojciechowski, Toni Ahtoniemi, Gundars Goldsteins, Raisa Giniatullina, Rashid Giniatullin, Jari Koistinaho and Johanna Magga
    Citation: Journal of Neuroinflammation 2011 8:74
  5. Experimental autoimmune encephalomyelitis (EAE) is a relevant animal model for the human demyelinating inflammatory disorder of the central nervous system (CNS), multiple sclerosis (MS). Induction of EAE by ad...

    Authors: Petra D Cravens, Rehana Z Hussain, Tresa E Zacharias, Li-Hong Ben, Emily Herndon, Ramya Vinnakota, Doris Lambracht-Washington, Stefan Nessler, Scott S Zamvil, Todd N Eagar and Olaf Stüve
    Citation: Journal of Neuroinflammation 2011 8:73
  6. Inflammation may be involved in the pathogenesis of Alzheimer's disease (AD). There has been little success with anti-inflammatory drugs in AD, while the promise of anti-inflammatory treatment is more evident ...

    Authors: J Couturier, M Paccalin, M Morel, F Terro, S Milin, R Pontcharraud, B Fauconneau and G Page
    Citation: Journal of Neuroinflammation 2011 8:72
  7. One of the most consistent biological findings in autism is the elevated blood serotonin levels. Immune abnormalities, including autoimmunity with production of brain specific auto-antibodies, are also commonl...

    Authors: Gehan Ahmed Mostafa and Laila Yousef AL-Ayadhi
    Citation: Journal of Neuroinflammation 2011 8:71
  8. How glial cells and cytokines are associated with the progression of delayed neuronal death induced by transient global ischemia is still unclear. To further clarify this point, we studied morphological change...

    Authors: Y Yasuda, T Shimoda, K Uno, N Tateishi, S Furuya, Y Tsuchihashi, Y Kawai, S Naruse and S Fujita
    Citation: Journal of Neuroinflammation 2011 8:70
  9. Amyotrophic lateral sclerosis (ALS) is a paralyzing disorder characterized by the progressive degeneration and death of motor neurons and occurs both as a sporadic and familial disease. Mutant SOD1 (mtSOD1) in...

    Authors: Eun Jin Yang, Seon Hwy Kim, Sun Choel Yang, Sang Min Lee and Sun-Mi Choi
    Citation: Journal of Neuroinflammation 2011 8:69
  10. The aim of the present study was to investigate microglia activation over time following traumatic brain injury (TBI) and to relate these findings to glutamate release.

    Authors: Hedy Folkersma, Jessica C Foster Dingley, Bart NM van Berckel, Annemieke Rozemuller, Ronald Boellaard, Marc C Huisman, Adriaan A Lammertsma, W Peter Vandertop and Carla FM Molthoff
    Citation: Journal of Neuroinflammation 2011 8:67
  11. Cerebral malaria (CM) is a lethal complication of Plasmodium falciparum infections. In the Plasmodium berghei ANKA (PbA) murine model, CM is associated with marked brain inflammation, increased expression of e...

    Authors: Graziela M Zanini, Pedro Cabrales, Wisam Barkho, John A Frangos and Leonardo JM Carvalho
    Citation: Journal of Neuroinflammation 2011 8:66
  12. The progranulin gene (PGRN) encodes a pleiotropic molecule with anti-inflammatory actions and neuronal protective effects. Accordingly, PGRN-deficient mice have been demonstrated to develop enhanced inflammation ...

    Authors: Paola Bossù, Francesca Salani, Antonella Alberici, Silvana Archetti, Giuseppe Bellelli, Daniela Galimberti, Elio Scarpini, Gianfranco Spalletta, Carlo Caltagirone, Alessandro Padovani and Barbara Borroni
    Citation: Journal of Neuroinflammation 2011 8:65
  13. The rising epidemic of obesity is associated with cognitive decline and is considered as one of the major risk factors for neurodegenerative diseases. Neuroinflammation is a critical component in the progressi...

    Authors: Pratibha V Nerurkar, Lisa M Johns, Lance M Buesa, Gideon Kipyakwai, Esther Volper, Ryuei Sato, Pranjal Shah, Domonkos Feher, Philip G Williams and Vivek R Nerurkar
    Citation: Journal of Neuroinflammation 2011 8:64
  14. Glutamate excitotoxicity contributes to oligodendrocyte and tissue damage in multiple sclerosis (MS). Intriguingly, glutamate level in plasma and cerebrospinal fluid of MS patients is elevated, a feature which...

    Authors: Olatz Pampliega, María Domercq, Federico N Soria, Pablo Villoslada, Alfredo Rodríguez-Antigüedad and Carlos Matute
    Citation: Journal of Neuroinflammation 2011 8:63
  15. The release of tumor necrosis factor-α (TNF-α) appears depend on the P2X7 receptor, a purinergic receptor. In the present study, we addressed the question of whether P2X7 receptor-mediated TNF-α regulation is ...

    Authors: Ji-Eun Kim, Hea Jin Ryu and Tae-Cheon Kang
    Citation: Journal of Neuroinflammation 2011 8:62
  16. By neutralizing the effect of the matrix metalloproteinases (MMPs), the tissue inhibitors of matrix metalloproteinases (TIMPs) play a critical role in maintaining tissue proteolysis in balance. As the major re...

    Authors: Jennifer V Welser-Alves, Stephen J Crocker and Richard Milner
    Citation: Journal of Neuroinflammation 2011 8:61
  17. Stroke is an important cause of morbidity and mortality and few therapies exist thus far. Mild hypothermia (33°C) is a promising neuroprotective strategy to improve outcome after ischemic stroke. However, its ...

    Authors: An-Gaëlle Ceulemans, Tine Zgavc, Ron Kooijman, Said Hachimi-Idrissi, Sophie Sarre and Yvette Michotte
    Citation: Journal of Neuroinflammation 2011 8:60
  18. Experimental autoimmune encephalomyelitis (EAE) is - in certain aspects - regarded as an animal model of the human CNS autoimmune disease multiple sclerosis (MS). While in EAE CNS-autoantigen-specific immunity...

    Authors: Vsevolod Smolianov, Thomas Dehmel, Patrick Vollmar, Anne K Mausberg, Bernd C Kieseier, Bernhard Hemmer, Hans P Hartung and Harald H Hofstetter
    Citation: Journal of Neuroinflammation 2011 8:59
  19. Macrophages play an important role in neuroinflammatory diseases such as multiple sclerosis (MS) and spinal cord injury (SCI), being involved in both damage and repair. The divergent effects of macrophages mig...

    Authors: Elly JF Vereyken, Priscilla DAM Heijnen, Wia Baron, Elga HE de Vries, Christine D Dijkstra and Charlotte E Teunissen
    Citation: Journal of Neuroinflammation 2011 8:58
  20. Kainic acid (KA)-induced status epilepticus (SE) was involved with release of free radicals. Sesamin is a well-known antioxidant from sesame seeds and it scavenges free radicals in several brain injury models....

    Authors: Peiyuan F Hsieh, Chien-Wei Hou, Pei-Wun Yao, Szu-Pei Wu, Yu-Fen Peng, Mei-Lin Shen, Ching-Huei Lin, Ya-Yun Chao, Ming-Hong Chang and Kee-Ching Jeng
    Citation: Journal of Neuroinflammation 2011 8:57
  21. Preterm and term newborns are at high risk of brain damage as well as subsequent cerebral palsy and learning disabilities. Indeed, hypoxia-ischemia (HI), pathogen exposures, and associated intracerebral increa...

    Authors: Marie-Elsa Brochu, Sylvie Girard, Karine Lavoie and Guillaume Sébire
    Citation: Journal of Neuroinflammation 2011 8:55
  22. Interleukin (IL)-6 is produced in the brain during peripheral infection and plays an important but poorly understood role in sickness behavior. Therefore, this study investigated the capacity of soluble gp130 ...

    Authors: Michael D Burton, Nathan L Sparkman and Rodney W Johnson
    Citation: Journal of Neuroinflammation 2011 8:54
  23. 3,4-Methylenedioxymethamphetamine (MDMA) produces a neuroinflammatory reaction in rat brain characterized by an increase in interleukin-1 beta (IL-1β) and microglial activation. The CB2 receptor agonist JWH-01...

    Authors: Elisa Torres, Maria D Gutierrez-Lopez, Andrea Mayado, Ana Rubio, Esther O'Shea and Maria I Colado
    Citation: Journal of Neuroinflammation 2011 8:53
  24. Although the cellular mechanisms responsible for the pathogenesis of autism are not understood, a growing number of studies have suggested that localized inflammation of the central nervous system (CNS) may co...

    Authors: Hongen Wei, Hua Zou, Ashfaq M Sheikh, Mazhar Malik, Carl Dobkin, W Ted Brown and Xiaohong Li
    Citation: Journal of Neuroinflammation 2011 8:52
  25. A critical point during the course of bacterial meningitis is the excessive influx of polymorphnuclear neutrophils (PMNs) from the blood into the brain. Both paracellular and transcellular routes of leukocyte ...

    Authors: Corinna Wewer, Annette Seibt, Hartwig Wolburg, Lilo Greune, M Alexander Schmidt, Jürgen Berger, Hans-Joachim Galla, Ulrike Quitsch, Christian Schwerk, Horst Schroten and Tobias Tenenbaum
    Citation: Journal of Neuroinflammation 2011 8:51
  26. Chronic neurodegeneration comprises an inflammatory response but its contribution to the progression of disease remains unclear. We have previously shown that microglial cells are primed by chronic neurodegene...

    Authors: Carol L Murray, Donal T Skelly and Colm Cunningham
    Citation: Journal of Neuroinflammation 2011 8:50
  27. Axl, together with Tyro3 and Mer, constitute the TAM family of receptor tyrosine kinases. In the nervous system, Axl and its ligand Growth-arrest-specific protein 6 (Gas6) are expressed on multiple cell types....

    Authors: Jason G Weinger, Celia F Brosnan, Olivier Loudig, Michael F Goldberg, Fernando Macian, Heather A Arnett, Anne L Prieto, Vladislav Tsiperson and Bridget Shafit-Zagardo
    Citation: Journal of Neuroinflammation 2011 8:49
  28. Human immunodeficiency virus type 1 (HIV-1) infection frequently causes neurologic disease, which is the result of viral replication and activation of macrophages and microglia in the CNS, and subsequent secre...

    Authors: Shengbo Cao, Chengxiang Wu, Yongbo Yang, Lynn F Sniderhan, Sanjay B Maggirwar, Stephen Dewhurst and Yuanan Lu
    Citation: Journal of Neuroinflammation 2011 8:48
  29. Vascular endothelial growth factor-A (VEGF-A) and chemokne ligand-2 (CCL2) levels have been examined in Amyotrophic Lateral Sclerosis (ALS) patients in Western countries. We measured these values in North Indi...

    Authors: Pawan K Gupta, Sudesh Prabhakar, Suresh Sharma and Akshay Anand
    Citation: Journal of Neuroinflammation 2011 8:47
  30. Tuberculosis (TB) of the central nervous system (CNS) is a deadly disease characterized by extensive tissue destruction, driven by molecules such as Matrix Metalloproteinase-2 (MMP-2) which targets CNS-specifi...

    Authors: Justin A Green, Shruti Dholakia, Karolina Janczar, Catherine WM Ong, Rachel Moores, Julie Fry, Paul T Elkington, Federico Roncaroli and Jon S Friedland
    Citation: Journal of Neuroinflammation 2011 8:46
  31. Hypoxic-ischemic (HI) brain injury remains a major problem in newborns, resulting in increased risk of neurological disorders. Neonatal HI triggers a broad inflammatory reaction in the brain, including activat...

    Authors: Linnea Stridh, Peter LP Smith, Andrew S Naylor, Xiaoyang Wang and Carina Mallard
    Citation: Journal of Neuroinflammation 2011 8:45
  32. We previously reported a correlation between levels of microparticles carrying CD31 (PMP CD31+) and disease activity in MS. However, the effects of long term (12 month) treatment with high dose, high frequency in...

    Authors: Mary Lowery-Nordberg, Erin Eaton, Eduardo Gonzalez-Toledo, Meghan K Harris, Kathrine Chalamidas, Jeanie McGee-Brown, Chaitanya V Ganta, Alireza Minagar, David Cousineau and J Steven Alexander
    Citation: Journal of Neuroinflammation 2011 8:43
  33. Traumatic brain injury (TBI) causes acute inflammatory responses that result in an enduring cascade of secondary neuronal loss and behavioral impairments. It has been reported that progesterone (PROG) can inhi...

    Authors: Fang Hua, Jun Wang, Tauheed Ishrat, Wenjing Wei, Fahim Atif, Iqbal Sayeed and Donald G Stein
    Citation: Journal of Neuroinflammation 2011 8:42
  34. Exposure to the nerve agent soman (GD) causes neuronal cell death and impaired behavioral function dependent on the induction of status epilepticus (SE). Little is known about the maturation of this pathologic...

    Authors: Erik A Johnson, Thuy L Dao, Michelle A Guignet, Claire E Geddes, Andrew I Koemeter-Cox and Robert K Kan
    Citation: Journal of Neuroinflammation 2011 8:41
  35. Apoptosis, neuroinflammation and blood-brain barrier (BBB) damage affect the susceptibility of the developing brain to hypoxic-ischemic (HI) insults. c-Jun N-terminal kinase (JNK) is an important mediator of i...

    Authors: Yi-Fang Tu, Yau-Sheng Tsai, Lan-Wan Wang, Hsin-Chieh Wu, Chao-Ching Huang and Chien-Jung Ho
    Citation: Journal of Neuroinflammation 2011 8:40
  36. Autoimmunity to the central nervous system (CNS) may play a pathogenic role in a subgroup of patients with autism. This study aimed to investigate the frequency of serum anti-ganglioside M1 auto-antibodies, as...

    Authors: Gehan A Mostafa and Laila Y AL-ayadhi
    Citation: Journal of Neuroinflammation 2011 8:39
  37. Previous studies indicate that successful resolution of Lyme neuroborreliosis (NB) is associated with a strong T helper (Th) 1-type cytokine response in the cerebrospinal fluid (CSF) followed by a down-regulat...

    Authors: Anna J Henningsson, Ivar Tjernberg, Bo-Eric Malmvall, Pia Forsberg and Jan Ernerudh
    Citation: Journal of Neuroinflammation 2011 8:36
  38. The transcription factors CCAAT/enhancer binding proteins (C/EBP) α, β and δ have been shown to be expressed in brain and to be involved in regulation of inflammatory genes in concert with nuclear factor κB (N...

    Authors: Veronica Ramberg, Linda M Tracy, Malin Samuelsson, Lars NG Nilsson and Kerstin Iverfeldt
    Citation: Journal of Neuroinflammation 2011 8:34
  39. Neuroinflammation and nitroxidative stress are implicated in the pathophysiology of neuropathic pain. In view of both processes, microglial and astroglial activation in the spinal dorsal horn play a predominan...

    Authors: Julie V Berger, Ronald Deumens, Stéphanie Goursaud, Sabrina Schäfer, Patricia Lavand'homme, Elbert A Joosten and Emmanuel Hermans
    Citation: Journal of Neuroinflammation 2011 8:33
  40. Adaptation to hypoxia and consequent pro-inflammatory gene expression of prostate and breast carcinomas have been implicated in the progression toward cancer malignant phenotype. Only partial data are availabl...

    Authors: Marco Tafani, Maura Di Vito, Alessandro Frati, Laura Pellegrini, Elena De Santis, Giovanni Sette, Adriana Eramo, Patrizio Sale, Emanuela Mari, Antonio Santoro, Antonino Raco, Maurizio Salvati, Ruggero De Maria and Matteo A Russo
    Citation: Journal of Neuroinflammation 2011 8:32
  41. Permanent functional deficits following spinal cord injury (SCI) arise both from mechanical injury and from secondary tissue reactions involving inflammation. Enhanced release of adenosine and glutamate soon a...

    Authors: Irene Paterniti, Alessia Melani, Sara Cipriani, Francesca Corti, Tommaso Mello, Emanuela Mazzon, Emanuela Esposito, Placido Bramanti, Salvatore Cuzzocrea and Felicita Pedata
    Citation: Journal of Neuroinflammation 2011 8:31
  42. Changes in the gene expressions for tumor necrosis factor-α (TNF-α) and/or interleukin-1β (IL-1β) during tinnitus have not been previously reported. We evaluated tinnitus and mRNA expression levels of TNF-α, I...

    Authors: Juen-Haur Hwang, Jin-Cherng Chen, Shan-Ying Yang, Ming-Fu Wang and Yin-Ching Chan
    Citation: Journal of Neuroinflammation 2011 8:30
  43. Status epilepticus (SE) is proposed to lead to an age-dependent acute activation of a repertoire of inflammatory processes, which may contribute to neuronal damage in the hippocampus. The extent and temporal p...

    Authors: Juha T Järvelä, Francisco R Lopez-Picon, Anna Plysjuk, Saku Ruohonen and Irma E Holopainen
    Citation: Journal of Neuroinflammation 2011 8:29

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